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These observations are consistent with the ideas that SHMT2 and MTHFD2 are key EBV-induced mediators of NADPH and glutathione production and that a significant role exists for mitochondrial 1C in newly infected cell redox defense
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Corticosteroids can: Reduce resistance to new infections Exacerbate existing infections Increase the risk of disseminated infections Increase the risk of reactivation or exacerbation of latent infections Mask some signs of infection Corticosteroid-associated infections can be mild but can be severe and at times fatal
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