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Diagnosing symptomatic AD in people with DS is quite complex because early symptoms can be mistaken as part of the individual’s lifelong intellectual disability or masked by coexisting medical comorbidities that might affect cognition, such as obstructive sleep apnea, hypothyroidism, and depression (456)
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In addition, ABCB1, ABCC1 and ABCC5 transporters might be involved in acetaminophen excretion in humans, as evident by changes in their expression after toxic acetaminophen ingestion [53]
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